Eat Fat, Get Thin: Why the Fat We Eat Is the Key to Sustained Weight Loss and Vibrant Health: 5 (The Dr. Hyman Library)

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Claims in the book were typically not well supported by their own references or by the scientific literature at large. Eat Fat, Get Thin’s references received a score of 1, indicating poor support for the book’s claims. The majority of the studies cited by the book do not support its claims. In some cases Eat Fat, Get Thin appears to mischaracterize, overstate, or withold elements of the studies that are cited. Criterion 1.3. How well does the strength of the claim line up with the strength of the evidence? We’ve been told lots of falsehoods about fat over the past 50 years that have shaped what we eat, what we buy, how we diet—all of which has had huge and disastrous consequences for our health. It all began with two big ideas about fat that have turned out to be wrong…The first was that all calories operate the same way in your body. Since fat has more than twice as many calories as carbs or protein per gram, the natural conclusion was that if you ate less fast, you would lose weight. That, in effect, the fat you ate turned to fat in your body. The second idea was that since fatty cholesterol deposits caused heart disease, and dietary fat, especially saturated fat, raised cholesterol, then the fat we ate caused heart disease. Seems to make sense, except the body is more complex than this simplistic conclusion would suggest…I’ll explain how the government and the food industry jumped on the bandwagon to create a maelstrom of bad advice. It was the perfect storm of overzealous scientists leaping to premature conclusions, anxious government agencies eager to do something to stem the tide of obesity and heart disease, and a profit-hungry food industry that raced in to capitalize on the low-fat mania, leading, in fact, to a rise in obesity, heart disease, and diabetes.” Ch2

Eat Fat, Get Thin: Why the Fat We Eat Is the Key to Sus… Eat Fat, Get Thin: Why the Fat We Eat Is the Key to Sus…

Taubes says as long as you avoid carbs you can eat all the protein and fat you want and not gain weight. Miller goes even further, claiming that a high fat diet will make you lose weight. I think any influence on weight loss is due to a decrease in calorie intake; admittedly, the satiety from fat might reduce total calorie consumption, but this has not been proven to have any practical effect on dieters.

I almost wish that Eat Fat, Get Thin had been divided into two books. One book presenting the historical overview, the scientific research, and the essential philosophy behind the concept of eating fat to lose weight. The other book presenting his 21 day weight-loss plan. The first book which I imagine consisting of Part I and Part II (How Did We Get Into This Big, Fat Mess? and Separating Fat From Fiction), I would have given three stars. The second book which I imagine consisting of Part III and Part IV (The Eat Fat, Get Thin Plan and Eat Fat, Get Thin Cooking and Recipes), I would have given one star--or perhaps two--if I'm generous.

Dr. Hyman’s Green Breakfast Smoothie - Dr. Mark Hyman Dr. Hyman’s Green Breakfast Smoothie - Dr. Mark Hyman

I read a lot of books like this. I enjoy the research and and posture the authors adopt. They all think their book is the definitive answer to what ails people. But putting that aside, I also love the research that is coming out and how this area is constantly evolving to a better understanding on how the body functions and what it needs to stay healthy. I grew up on the four basic food groups and the food pyramid that replaced that. I grew up thinking eating fat was bad, so I find that where modern research is taking us now, is kind of fascinating. Update 10 monthe later, I continue to eat this way and continue to feel better. I have lost 6 more lbs. Slower than the first 30 but I think I am finding my new normal. One thing I am very happy about is that after being a life long allergy sufferer I only have small reactions now and only to pollen in Spring and fall or to perfumes. I am now able to eat dairy with little or no reactions. I do try to do only grass fed dairy but not being real strict and happy to have some dairy now! is it meat vs veggies, or rather the sugar and refined carbs that are part of the typical meat eater’s diet that we should be concerned with?He approves delicious meats! Loads of veggies, fruits, nuts, potatoes, and all the brain-feeding fats such as ghee, butter, avo, and various other *good* oils. Canola, you’re not welcome here. When we looked for more recent studies and meta-analyses related to prostate cancer we found conflicting evidence. For example, a 2015 meta-analysis found no link between total fat and prostate cancer. However, in a more recent population level study published in 2019, known as the SABOR study, a relationship was observed between both total fat intake and saturated fat and the risk of developing prostate cancer. Although we do note that this study was published after Eat Fat, Get Thin was released. Another recent paper reviewed the current state of the literature and also highlighted that there is some disagreement in the literature. They found that most preclinical trials show a relationship between fat and prostate cancer while only some human trials demonstrate this relationship. They do however note that dietary patterns that are low in saturated fat (specifically, the Mediterranean diet and the DASH diet) appear to have more reliable evidence for their efficacy in protecting against developing prostate cancer. Overall we found that for prostate cancer the connection with fat does appear to be conflicting as Eat Fat, Get Thin suggests. Small portions of "starchy" veggies (1/2 cup to 1 cup at a time, but, only 4 times a week) This includes beets, celeriac, parsnips, pumpkin, rutabaga, sweet potatoes, turnips, winter squash. I am one of those “my tummy always hurts!” girls, and this book my just well have changed my entire perspective on food. I feel great for the first time in forever, and I have Dr. Mark to thank. I read this right after Always Hungry by Dr. David Ludwig. They both cover similar ground (the food industry is more concerned with profit than creating nutritious foods; the food industry gives lots of money to our politicians-- which is why they aren't doing a better job of creating better food regulations; confusion about the complex science of nutrition have led to bad recommendations over the years-- which is why consumers are confused, to whit: fat isn't as bad for you as you think it is). They both include lots of cited research and cover the science of nutrition. They both recommend increased dietary intake of certain "good" fats, more non-starchy vegetables, reduced carbs, and very little sugar). Dr. Hyman's plan is a little more hard-core and, I think, somewhat unrealistic for: poor people, working people, and/or beginners. I think Dr. Ludwig's approach might be a little more manageable.

Eat Fat, Get Thin? | Science-Based Medicine Eat Fat, Get Thin? | Science-Based Medicine

He has commercialized himself and, as with all such television-doctor marketing superstars, I would recommend they all read "The Citadel" by Cronin, about a doctor who became a dedicated physician to help the destitute in England, who veered off course and hawked useless remedies to rich people for personal profit, at the possible loss of his soul and loss of original humanitarian aspiration. I've seen some pretty irresponsible, even dangerous, recommendations on these television shows including PBS, and they all cover their butts by either saying it's either for entertainment value, or that they should always consult their own doctor first. Coconut oil is extracted from the dried flesh of the coconut. Coconut oil is made up of 86 percent saturated fat, 6 percent monounsaturated fat, and 1.4 percent polyunsaturated fat. About half of the saturated fat in coconut oil is a rare, special type of saturated fat called lauric acid…In the body, lauric acid converts to monolaurin, one of the compounds found in breast milk that boost a baby’s immune system…It is like superfuel for your cells, your metabolism, your bones, and your brain…The saturated fat in coconut oil is aa very rate, very beneficial type called medium-chain triglycerides (MCT). These saturated fats actually reduce the ratio of total cholesterol to HDL and promote weight loss and can even heal fatty lover caused by obesity…In the body, MCTs convert easily into energy; therefore, very little MCT oil is stored as fat…This is how MCTs help you burn fat and lose weight…Look for coconut oil that is virgin, organic, cold-pressed, unrefined, and never deodorized or bleached. You can use expeller-pressed, unrefined coconut oil for cooking at up to 400F, so this is a go-to oil for high-heat stir fries, medium-high heat sautéing, and most baking.” -Ch8

I care as much as Dr. Hyman does, so I felt compelled to speak out. Was a certified Natural Therapeutics Specialist and Licensed Massage Therapist for more than twenty years, have a firm foundation. Also worked for physicians for about fourteen years and know how and what they think. I believe Hyman cares, but... I do have concerns. Guys like Chopra and Oz, with their new-age piecemeal unqualified advice outside their profession, worry me. This reference received a score of 3, indicating that it offers moderate support for the claim. In this study mice were placed onto 1 of 4 diets: a ketogenic diet (78.9% fat, 9.5% protein, and 0.76% carbohydrate), a typical chow diet (6.5% fat, 23.5% protein, and 56% carbohydrate), a high-fat, high-sugar “obesogenic diet” (24% fat, 24% protein, and 41% carbohydrate), and a calorie restricted diet (66% calorie restricted). The obesogenic diet group gained weight, the low-fat diet group appeared to maintain their weight, and the ketogenic and calorie restricted diet groups both lost about equivalent amounts of weight even though the keto group consumed more calories. It is clear that Eat Fat, Get Thin is trying to compare the keto group to the control group (which has a higher carb content than the keto group) as the quote from Eat Fat, Get Thin states “weight loss was greater than in rats fed a high-carb diet, even though they ate exactly the same number of calories”. While the results of the study do support the weight loss claim we do note that the mice on the low-carb diet lost their weight from the nonfat component of their weight, likely water weight. So, although there was a difference in weight between the control diet and the keto diet mice there was no difference in body fatness. Therefore, this study actually raises some questions as to how beneficial the reduction in weight from nonfat tissue is for health. We would also like to clarify that the cited paper reports on an experiment in mice, not rats as claimed by Eat Fat, Get Thin. Overall (average) score for reference accuracy

Eat Fat Get Thin: Why the Fat We Eat Is the Key to Sustained

The claim received a score of 2, indicating that the claim is moderately overstated. In some cases, such as with prostate cancer, there did appear to be conflicting evidence. However, for other types of cancer, such as breast cancer, the relationship with fat was more apparent. Overall (average) score for claim 3 it’s not eating more and exercising less that makes you fat – rather, BEING fat makes you eat more and exercise less Turning to prostate cancer, Eat Fat, Get Thin provides some weak epidemiological evidence derived from a single food frequency questionnaire that found that individuals who consume more dairy products are more likely to develop prostate cancer. The book also cites a review paper that summarizes the evidence for the relationship between dietary factors and prostate cancer. This paper reports a link between saturated fat and prostate cancer, although the association between total fat intake and prostate cancer risk is inconsistent. Eat Fat, Get Thin uses this paper to argue that intakes of omega-3 fatty acids have been shown to reduce the risk of prostate cancer, which the paper does state, but withholds that they also found that increased saturated fat intake was associated with a higher risk of prostate cancer. One other piece of evidence that Eat Fat, Get Thin provides for their argument is an opinion paper, which calls for restrained enthusiasm around the topic of fat intake and prostate cancer. The paper actually provides references to a large amount of preclinical and case-control data demonstrating a link between fat and prostate cancer while urging caution when looking at human data. Particularly it highlights conflicting data found in observational and prospective cohort data in humans. This paper is used to try to undermine the claims that fat is related to both prostate and breast cancer risk. However, this paper was published several years before the papers previously cited by Eat Fat, Get Thin. This means that this opinion piece was published prior to the publication of the human data showing stronger links between fat and prostate and breast cancer risk. The claim received a score of 1, indicating that the claim is greatly overstated. As noted previously, while there is epidemiological evidence to support the claim made by Eat Fat, Get Thin there is also much that disputes it. Stronger forms of evidence such as randomized controlled trials and metabolic ward studies are unsupportive of the claim made by the book. Additionally, dietary patterns low in saturated fat appear to be beneficial for both cholesterol and heart disease outcomes, particularly when compared to dietary patterns higher in saturated fat. Overall, the claim made by the book does not line up with the bulk of the scientific literature available. Overall (average) score for claim 2This reference received a score of 2, indicating that it offers weak support for the claim. The cited study reports on the extraction of polyphenols and lectins from a variety of beans, however, it does not report any data on the quantity of lectins that are contained within beans, how much remains after cooking, whether this quantity is concerning. Additionally, the paper is more heavily focused on the polyphenol content of the beans and the potential for polyphenols to agglutinate erythrocytes in cell cultures, but not in humans. Reference 8 Reference